肿瘤与衰老

doi:10.3760/cma.j.issn.1673-422X.2017.07.010

国际肿瘤学杂志 ›› 2017, Vol. 44 ›› Issue (7): 519-521.doi: 10.3760/cma.j.issn.1673-422X.2017.07.010

肿瘤与衰老

张百红，岳红云

730050兰州军区兰州总医院肿瘤科（张百红），眼科（岳红云）

收稿日期:2017-01-03 出版日期:2017-07-08 发布日期:2017-06-20
通讯作者: 张百红 E-mail:bhzhang1999@126.com
基金资助:
甘肃省自然科学基金（1308RJZA181）

Cancer and aging

Zhang Baihong, Yue Hongyun

Department of Oncology, Lanzhou General Hospital of Lanzhou Military Region, Lanzhou 730050, China

Received:2017-01-03 Online:2017-07-08 Published:2017-06-20
Contact: Zhang Baihong E-mail:bhzhang1999@126.com
Supported by:
Natural Science Foundation of Gansu Province of China (1308RJZA181)

摘要/Abstract

摘要： 肿瘤与衰老发生机制的相似性提示肿瘤可能是衰老的自然结果。通过线粒体置换、基因组编辑、端粒维护和免疫编辑可以诱导肿瘤细胞衰老或抵抗机体衰老，从而突破人类寿命的自然极限。

关键词: 肿瘤, 衰老, 免疫衰老

Abstract: The similar mechanism of human cancer and aging means that cancer is the natural result of aging. Mitochondrial replacement, genome editing, telomere synthesis and immune editing can induce cancer cell senescence and resist human aging, which may breach a limit to human lifespan.

张百红，岳红云. 肿瘤与衰老[J]. 国际肿瘤学杂志, 2017, 44(7): 519-521.

Zhang Baihong, Yue Hongyun. Cancer and aging[J]. Journal of International Oncology, 2017, 44(7): 519-521.

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肿瘤与衰老

Cancer and aging

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