[1] Guo L, Li N, Wang G, et al. Body mass index and cancer incidence: a prospective cohort study in northern China[J]. Zhonghua Liu Xing Bing Xue Za Zhi, 2014, 35(3): 231-236.
[2] Poorolajal J, Jenabi E, Masoumi SZ. Body mass index effects on risk of ovarian cancer: a metaanalysis[J]. Asian Pac J Cancer Prev, 2014, 15(18): 7665-7671.
[3] Olsen CM, Nagle CM, Whiteman DC, et al. Obesity and risk of ovarian cancer subtypes: evidence from the Ovarian Cancer Association Consortium[J]. Endocr Relat Cancer, 2013, 20(2): 251-262.
[4] Canchola AJ, Chang ET, Bernstein L, et al. Body size and the risk of ovarian cancer by hormone therapy use in the California Teachers Study cohort[J]. Cancer Causes Control, 2010, 21(12): 2241-2248.
[5] De Stefano I, Zannoni GF, Prisco MG, et al. Cytoplasmic expression of estrogen receptor beta (ERbeta) predicts poor clinical outcome in advanced serous ovarian cancer[J]. Gynecol Oncol, 2011, 122(3): 573-579.
[6] Lu Z, Zhang Y, Yan X, et al. Estrogen stimulates the invasion of ovarian cancer cells via activation of the PI3K/AKT pathway and regulation of its downstream targets Ecadherin and alphaactinin4[J]. Mol Med Rep, 2014, 10(5): 2433-2440.
[7] Tas F, Karabulut S, Serilmez M, et al. Clinical significance of serum insulinlike growth factor1 (IGF1) and insulinlike growth factor binding protein3 (IGFBP3) in patients with epithelial ovarian cancer[J]. Tumour Biol, 2014, 35(4): 3125-3132.
[8] Zhang Y, Daquinag AC, AmayaManzanares F, et al. Stromal progenitor cells from endogenous adipose tissue contribute to pericytes and adipocytes that populate the tumor microenvironment[J]. Cancer Res, 2012, 72(20): 5198-5208.
[9] Chen C, Chang YC, Lan MS, et al. Leptin stimulates ovarian cancer cell growth and inhibits apoptosis by increasing cyclin D1 and Mcl1 expression via the activation of the MEK/ERK1/2 and PI3K/Akt signaling pathways[J]. Int J Oncol, 2013, 42(3): 1113-1119.
[10] 陈仲. 脂联素蛋白的纯化方法及卵巢癌ES2体外抑瘤活性研究[D]. 北京: 中国协和医科大学, 2007.
[11] Dalamaga M. Resistin as a biomarker linking obesity and inflammation to cancer: potential clinical perspectives[J]. Biomark Med, 2014, 8(1): 107-118.
[12] Nieman KM, Kenny HA, Penicka CV, et al. Adipocytes promote ovarian cancer metastasis and provide energy for rapid tumor growth[J]. Nat Med, 2011, 17(11): 1498-1503.
[13] Fotopoulou C, Richter R, Braicu EI, et al. Impact of obesity on operative morbidity and clinical outcome in primary epithelial ovarian cancer after optimal primary tumor debulking[J]. Ann Surg Oncol, 2011, 18(9): 2629-2637.
[14] Worley MJ Jr, Guseh SH, RauhHain JA, et al. What is the optimal treatment for obese patients with advanced ovarian carcinoma?[J]. Am J Obstet Gynecol, 2014, 211(3): 231.e1-9.
[15] Barrett SV, Paul J, Hay A, et al. Does body mass index affect progressionfree or overall survival in patients with ovarian cancer? Results from SCOTROC Ⅰ trial[J]. Ann Oncol, 2008, 19(5): 898-902.
[16] Wright JD, Tian C, Mutch DG, et al. Carboplatin dosing in obese women with ovarian cancer: a Gynecologic Oncology Group study[J]. Gynecol Oncol, 2008, 109(3): 353-358.
[17] AuYeung G, Webb PM, DeFazio A, et al. Impact of obesity on chemotherapy dosing for women with advanced stage serous ovarian cancer in the Australian Ovarian Cancer Study (AOCS)[J]. Gynecol Oncol, 2014, 133(1): 16-22.
[18] Slaughter KN, Thai T, Penaroza S, et al. Measurements of adiposity as clinical biomarkers for firstline bevacizumabbased chemotherapy in epithelial ovarian cancer[J]. Gynecol Oncol, 2014, 133(1): 11-15. |